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Introduction

ECG effects of hypocalcemia

Rhythm

  • Regular

Rate

  • Within normal limits

P wave

  • Normal size
  • Normal configuration

PR interval

  • Within normal limits

QRS complex

  • Within normal limits

T wave

  • Normal size
  • Normal configuration
  • May be flat or inverted

QT interval

  • Prolonged from decreased calcium level (the key finding, as shown in shaded area on strip)

Other

  • Prolonged ST segment


What Causes It

  • Inadequate calcium intake due to diet deficient in green, leafy vegetables and dairy products
  • Excessive phosphorus intake (binds with calcium and prevents calcium absorption)
  • Pancreatitis (decreases ionized calcium)
  • Blood administration (the citrate solution in stored blood binds with calcium)
  • Neoplastic bone metastasis (decrease serum calcium levels)
  • Vitamin D deficiency due to inadequate intake or inadequate exposure to sunlight
  • Malabsorption of fats
  • Inadequate PTH levels (caused by removal of parathyroid glands)
  • Metabolic or respiratory alkalosis
  • Hypoalbuminemia (low albumin level)

What to Look for

  • Carpopedal spasm
  • Circumoral or digital paresthesia
  • Confusion
  • Hyperactive bowel sounds
  • Hyperreflexia
  • Intestinal cramping
  • Positive Chvostek sign
  • Positive Trousseau sign

Severe Hypocalcemia

  • Tetany
  • Seizures
  • Respiratory arrest
  • Death

How It's Treated

  • Identify and manage the underlying cause.
  • Monitor serum calcium levels.
  • Administer IV calcium gluconate for severe symptoms. Keep calcium gluconate on hand for a patient with a positive Trousseau or Chvostek sign. Hypocalcemia may progress quickly to tetany, seizures, respiratory arrest, and death.
  • Replace calcium orally.
  • Identify and manage cardiac arrhythmias.
  • Instruct the patient to decrease phosphate intake.