Also known as ventricular escape rhythm, they are the rhythm of last resort
Acts as safety mechanism to prevent ventricular standstill (asystole) when no impulses are conducted to the ventricles from above the bundle of His
The cells of the HisPurkinje system take over and act as the heart's pacemaker to generate electrical impulses with an inherent firing rate of usually 20 to 40 beats/min
They occur when all the heart's other pacemakers have failed or when supraventricular impulses can't reach the ventricles due to a block in the conduction system
When the rate of an ectopic pacemaker site in the ventricles is less than 100 beats/min but exceeds the inherent ventricular escape rate of 20 to 40 beats/min, the rhythm is referred to as accelerated idioventricular rhythm (it's usually related to enhanced automaticity of ventricular tissue and has the same ECG characteristics as idioventricular rhythm except for heart rate)
On the line
Identifying idioventricular rhythm
This rhythm strip illustrates idioventricular rhythm. Look for these distinguishing characteristics:
Rhythm
Regular
Rate
Unable to determine atrial rate; ventricular rate of 35 beats/min
P wave
Absent
PR interval
Unmeasurable
QRS complex
Wide and bizarre
T wave
Deflection opposite QRS complex
QT interval
0.60 second
Other
None
What Causes It⬆⬇
Digoxin toxicity
Drugs
Beta-adrenergic blockers
Calcium channel blockers
Tricyclic antidepressants
Failure of all of the heart's higher pacemakers
Failure of supraventricular impulses to reach the ventricles because of a block in the conduction system
An accelerated idioventricular rhythm has the same characteristics as an idioventricular rhythm except that it's faster. The rate shown here varies between 40 and 100 beats/min.
What to Look for⬆⬇
Evidence of sharply decreased cardiac output (such as hypotension, dizziness, feeling of faintness, light-headedness, and syncope) if the patient has a continuous idioventricular rhythm
Difficult auscultation or palpation of blood pressure
How It's Treated⬆
Treatment should begin immediately to increase heart rate, improve cardiac output, and establish a normal rhythm.
Treatment isn't intended to suppress the idioventricular rhythm because this arrhythmia acts as a safety mechanism against ventricular standstill.
Never treat an idioventricular rhythm with antiarrhythmic drugs (such as amiodarone or lidocaine) because these drugs suppress the escape beats.
Atropine may be given to increase heart rate. If atropine isn't effective or the patient develops hypotension or other evidence of clinical instability, a pacemaker may be inserted to reestablish a heart rate and cardiac output sufficient to perfuse organs.
A transcutaneous pacemaker may be used in an emergency until a transvenous pacemaker can be inserted.
Maintain continued ECG monitoring and periodically assess the patient until hemodynamic stability has been restored.
Keep atropine and pacemaker equipment readily available.
Enforce bed rest until an effective heart rate has been maintained and the patient is stable.
Tell the patient and his family about the serious nature of this arrhythmia and the treatment it requires.
If the patient needs a permanent pacemaker, explain how it works, how to recognize problems, when to contact a doctor, and how pacemaker function will be monitored.