A. Bicipital Tendinitis
- Symptoms
- Pain, usually in anterior portion of shoulder
- Patients can usually point to point of maximal focal tenderness
- Either (or both) heads of the biceps may be involved
- Probably due to impingement of biceps tendon by acromion
- May be coincident with rotator cuff tendinitis
- Treatment
- Rest
- Hot packs
- Physical Therapy
- Ultrasound Therapy
- Passive then active range of motion (ROM) exercises
- Non-steroidal antiinflammatory drugs (NSAIDs)
- Good acute response to lidocaine + DepoMedrol® followed by 1-2 months of NSAIDs
B. Rotator Cuff Tendinitis [6]
- Typically associated with calcification of tendons in rotator cuff
- This causes calcific tendonitis which is probably most common final step in pathology
- Also called Impingement Syndrome: supraspinatus tendon impinged on by acromion
- Osteophyte development, especially inferior aspect of acromioclavicular joint
- Characteristics
- Most common cause of shoulder pain
- Pain worse when arm is moved forward with elbow flexed
- Active motion worse than passive motion
- Underlying Causes
- Probably multifactorial
- Overuse most common contributing factor, usually with overhead reaching
- Acute shoulder trauma
- History of inflammatory joint disease, usually with shoulder symptoms
- Joint diseases include rheumatoid arthritis (RA), pseudogout, gout
- Diagnosis with shoulder magnetic resonance imaging (MRI)
- Treatment
- Conservative care with rest, hot or cold packs, ± NSAIDs
- Physical therapy
- Manipulative therapy for shoulder girdle symtoms can accelerate recovery [4]
- Resistant cases can be treated with glucocorticoids or ultrasound therapies
- Lidocaine injection usually into subacromial bursa can be diagnostic and therapeutic
- If lidocaine improves symptoms, then ~20mg of Depo-Medrol can be infused as well
- Ultrasound - 24 x 15 minute treatments improves pain and reduces calcification [1]
- Low or high dose ultrasound (electrocorporeal shock wave therapy) highly effective [2]
- NSAIDs
C. Rotator Cuff Tear [2,6]
- Roator-cuff tendon failure due to either tear or wear is most common clinical shoulder problem
- Anatomy of the Rotator Cuff [6]
- Rotator cuff is the integration of the capsule of the glenohumeral joint with tendons derived from four different muscles
- Tendons arise from supraspinatus, infraspinatus, teres minor, and subscapularis ("SITS")
- Biceps tendons (two heads, medial and lateral) run just under the rotator cuff
- The individual muscles of the cuff help provide strength in arm movement
- Subscapularis aides in internal rotation
- Supraspinatus muscle aides in elevation
- Infraspinatus and teres minor muscles aid in external rotation
- Classification of Tear
- Acute or Chronic
- Partial or Complete
- Causes
- <50% are associated with trauma
- The majority of the remainder are due to age-related attrition of the tendons
- Any disease process which weakens the tendons can contribute to dysfunction
- Chronic inflammatory diseases: especially RA (rheumatoid arthritis), pseudogout (CPPD)
- Cuff defects more common in obese persons
- Glucocorticoid injections do not increase risk of cuff failure, but may alter tendon composition
- Smoking (nicotine) may reduce ability of tendons to heal and attach to bones
- Symptoms
- Shoulder pain ± crepitus on active movement
- Weakness on abduction
- Inability to maintain 90° of passive abduction indicates large tear in 98% of cases [3]
- Patients with complete tear may have superiorly migrated shoulders
- Radiograph should generally be done to rule out fractures
- High Clinical Suspicion for Tear
- Supraspinatus weakness
- Weakness in external rotation
- Impingement sign (pain)
- Presence of all three signs: 98% probability of tear
- Presence of two of the three signs AND age >60: 98% probability of tear
- Absence of any of the signs: <5% chance of tear
- Diagnosis
- Shoulder films usually not helpful except to show fractures, RA changes or chondrocalcinosis
- MRI and ultrasound are similarly accurate, 97% positive predictive values, >67% specificity [6]
- Definitive diagnosis by arthrogram with computerized tomography (CT) or open surgery
- This should show communication between glenohumeral joint and subacromial bursa
- Small ulcer-like crater in rotator cuff may be seen in incomplete tears
- Treatment
- Large tears, especially in young persons, should be surgically repaired
- Acute cuff failure should be treated rapidly (as with any rupture of major tendon)
- Prolonged observation / non-surgical management allow tendon to retract and muscle atrophy
- Chronic, full-thikcness degenerative tendon defects best managed without surgery
- Small complete and larger incomplete tears are treated conservatively
- This includes NSAIDs, rest, and physical therapy (stretching and muscle strengthening)
- Subacromial injections of lidocaine/steroids are usually no better than NSAIDS at relieving pain
- Local injection is reasonable for NSAID failures or intolerance
- Repeated glucocorticoid injections are not recommended
- Surgical Repair
- For acute, full tendon ruptures, surgery is indicated
- For more chronic situations, surgery is considered if symptoms persist
- Surgery goal is to reattached the detached cuff tendon
- Surgery more likely to be effective if age <60 years, traumatic onset of weekness, symptoms for <2 months, <4 glucocorticoid injections, no previous shoulder surgery or muscle atrophy
- Surgical risks include infection, postsurgical adhesions with loss of motion, deltoid injury
- Injury to axillary nerve, roughness in humeroscapular motion, damage to coaracoacromial arch also occur
D. Adhesive Capsulitis
- Introduction
- Also called "frozen shoulder" or pericapsulitis
- Rare prior to age 40
- May occur secondary to any type of shoulder problem and is usually chronic
- Diabetes and inflammatory shoulder arthritis may predispose
- Failure to use shoulder and/or low pain threshhold may exacerbate disease
- Symptoms
- Generalized pain and tenderness with severe loss of active and passive motion
- Muscle atrophy may occur early in the course
- Pathology
- Formation of fibrous adhesions between joint capsule and humeral neck
- Axillary fold binds to itself causing restriction of joint motion
- Capsule becomes thickened and contracted
- Diagnosis
- Clinical symptoms with physical findings are fairly accurate
- In severe cases, arthrography can be used
- Arthrogram shows decrease in infusible dye (<10mL; normal ~30mL)
- Treatment
- NSAIDs are usually suboptimal
- Subacromial ± glenohumeral lidocaine+DepoMedrol (20-40mg) injections
- Physical therapy with ice packs, ultrasound, TENS, gentle ROM exercises
- ROM exercises include pendulum (arm swinging) and wall climbing (with fingers) motions
E. Shoulder Instability and Dislocation [5]
- Instability of the glenohumeral joint occurs when shoulder structures provide too little stability when humerus moves on the glenoid; pain is the usual result
- Result is that head of the humerus fails to stay in the glenoid fossa on motion
- Tears in the labrum (cartilage rim of glenoid) can contribute to pain
- Subluxation is symptomatic translation of the humeral head without complete separation
- Dislocation: humeral head has no attachment to glenoid fossa (complete separation)
- Three Causes of Instability
- Anterior luxation occurs with sudden fall with an outstreched arm (skiers) or with blocked throwing motion, usually reduced in the field
- Chronic luxation due to chronic gradual overhead stretching, without obvious trauma
- Hyperlaxicity of glenohumeral capsule, usually congenital without trauma, less common
- Shoulder Dislocation
- Very painful condition
- Patient will hold arm in fixed position
- Shoulder Instability (without Dislocation)
- More subtle presentation than dislocation
- Dead arm feeling
- Pain and functional disability nonspecific for presence of instability
- Physical exam should attempt to evoke symptoms
- Evaluate for presence of glenohumeral joint laxity
- Relocation test and anterior release test are best for establishing diagnosis of instability or intra-articular pathology
- Relocation test performed to relieve symptoms (pain and apprehension) of instability
- Relocation test conducted with patient supine and the arm aaabducted to 90° and extrenally rotated to 90°; apply downward pressure to humeral head
- Anterior release test conduct similar to relocation test, then examiner's hand is removed suddenly releaseing pressure on the humeral head
References
- Eisenbichler GR, Erdogmus CB, Resch KL, et al. 1999. NEJM. 340(2):1533
- Gerdesmeyer L, Wagenpfeil S, haake M, et al. 2003. JAMA. 290(19):2573
- Murrell GAC and Walton JR. 2001. Lancet. 357(9258):769
- Bergman GJD, Winters JC, Groenier KH, et al. 2004. Ann Intern Med. 141(6):432
- Luime JJ, Verhagen AP, Miedema HS, et al. 2004. JAMA. 292(16):1989
- Matsen FA III. 2008. NEJM. 358(20):2138