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Information

Bone and Soft-Tissue Disorders

= excess iron deposition in tissues (hemosiderosis) resulting in tissue injury

Primary / Idiopathic Hemochromatosis!!navigator!!

= autosomal recessive genetic disorder (abnormal iron-loading gene on short arm of chromosome 6) in thalassemia, sideroblastic anemia

Defect: increased absorption + parenchymal accumulation of dietary iron

Organs: liver, pancreas, heart (parenchymal iron overload)

Homozygous frequency: 1÷200; M÷F = 1÷1

Age:>30 years (M), usually after menopause (F);

◊Females protected by menstruation

  • cirrhosis (frequently present at time of diagnosis)
  • “bronzed diabetes” (50%) = insulin-dependent diabetes ← excess intracellular iron reduces function of beta islet cells while reserve capacity of exocrine function is not exceeded by toxic effects of iron; skin pigmentation
  • congestive cardiomyopathy ← myocardial muscle accumulates toxic levels of intracellular iron
  • hypogonadism + decreased libido ← pituitary dysfunction
  • slowly progressive arthritic symptoms (30%)
  • increased serum iron + ferritin level (nonspecific)
  • transferrin saturation + serum ferritin assay (specific)
  • Skeleton
    Distribution: most commonly in hands (metacarpal heads, particularly 2nd + 3rd MCP joints), carpal (30–50%) + proximal interphalangeal joints, knees, hips, elbows
    • generalized osteoporosis
    • small subchondral cystlike rarefactions with fine rim of sclerosis (metacarpal heads)
    • arthropathy in 50% (iron deposition in synovium)
    • uniform symmetric joint space narrowing (unusual for degenerative joint disease)
    • enlargement of metacarpal heads:
      • hook-like osteophytes on radial aspect of metacarpal heads (CHARACTERISTIC)
    • chondrocalcinosis in >60%, knees most commonly affected
      1. calcium pyrophosphate deposition ← inhibition of pyrophosphatase enzyme within cartilage which hydrolyzes pyrophosphate to soluble orthophosphate
      2. calcification of triangular cartilage of wrist, menisci, annulus fibrosus, ligamentum flavum, symphysis pubis, Achilles tendon, plantar fascia
  • Brain
    • marked loss in SI of anterior lobe of pituitary gland ← iron deposition
  • Abdomen
    • decreased T2 signal intensity in liver, pancreas
    • normal SI of spleen on T2WI + T2*WI

Dx: liver biopsy with hepatic iron index >2 (= iron concentration in µmol/g of dry weight divided by patient's age in years)

Cx: hepatoma in 14% (iron stimulates growth of neoplasms)

Prognosis: death from CHF (30%), death from hepatic failure (25%), death from HCC (in up to 33%)

Rx:

  1. Phlebotomy (returns life expectancy to normal if instituted prior to complications)
  2. Screening of family members (gene can be located by human leukocyte antigen typing)

DDx:

  1. Pseudogout (no arthropathy)
  2. Psoriatic arthritis (skin + nail changes)
  3. Osteoarthritis (predominantly distal joints in hands)
  4. Rheumatoid arthritis
  5. Gout (may also have chondrocalcinosis)

Secondary Hemochromatosis!!navigator!!

= nongenetic iron overload

  1. ineffective erythropoiesis: thalassemia major increases demand for iron resulting in increased absorption + retention of dietary iron
    Cx: increase in parenchymal iron
  2. Bantu siderosis = parenchymal + RES iron overload ← extensive use of iron pots for cooking

Transfusional Siderosis!!navigator!!

[siderosis = exposure to excess iron]

= iron overload of RES (NOT a form of secondary hemochromatosis); iron less toxic

Pathophysiology: large number of damaged erythrocytes from blood transfusions are incorporated into RES cells + undergo lysis with liberation of iron from hemoglobin (extravascular hemolysis)

Organs of RES: Kupffer cells, spleen, bone marrow

  • decreased signal intensity of liver + spleen on T2WI

Iron storage capacity of RES: 10 g (equivalent to 40 units of packed RBCs)


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