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Table 52-1

Etiology
Hemorrhage or Extensive Tissue InjuryCardiac TamponadeMyocardial ContusionPneumothorax or HemothoraxSpinal Cord InjurySepsis
Primary mechanismsHypovolemiaVentricular inflow restrictionDiminished ventricular performance and elevated pulmonary vascular resistanceLung collapse
Mediastinal shift, causing inflow and outflow obstruction of the heart
Vasodilation and relative hypovolemia caused by loss of sympathetic toneIntestinal perforation causing peritoneal contamination
Typical signs and symptomsTachycardia
Narrow pulse pressure
Cold, clammy skin from vasoconstriction
Tachycardia
Hypotension
Dilated and engorged neck veins
Muffled heart sounds
Diminished BP response to fluid challenge
Dysrhythmia
Tachycardia
Hypotension
Tachycardia
Hypotension
Dilated and engorged neck veins
Absent breath sounds
Hyperresonance to percussion
Tracheal shift
Dyspnea
Subcutaneous emphysema
Hypotension without tachycardia, cutaneous vasoconstriction, or narrow pulse pressureDevelops mainly a few hours after colon injury
In hypovolemic patients, signs and symptoms indistinguishable from hypovolemic shock
In normovolemic patients, fever, modest tachycardia; warm, pink skin; near normal BP; wide pulse pressure
Hypotension may develop
Treatment continuum, from least to most intenseCrystalloids initially
Transfusion if 2,000 mL of crystalloid in 15 min does not restore BP
Pericardiocentesis
Pericardial window
ED thoracotomy
Fluids
Fluids and vasodilators
Fluids and inotropes
Release of air with 14-gauge catheter
Chest tube
Fluids
Fluids and vasopressors
Fluids, vasopressors, and inotropes, if myocardial damage is present
Fluids and antibiotics
Fluids, antibiotics, and inotropes for hypotension

BP = blood pressure; ED = emergency department.

Reproduced with permission from American College of Surgeons, Committee on Trauma. Shock, Advanced Trauma Life Support Student Course Manual. Edited by the American College of Surgeons, 8th ed.Chicago: American College of Surgeons; 2008:55–71.