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Evidence summaries

Dabigatran Versus Vitamin K Antagonists in Non-Valvular Atrial Fibrillation

Dabigatran 150 mg twice daily is marginally more effective in the prevention of vascular deaths and ischaemic events than treatment with dose-adjusted warfarin in patients with atrial fibrillation. Dabigatran may be associated with a lower risk of intracranial bleeding, but it may increase the risk of gastrointestinal bleeding, especially in those aged 75 years and older, compared to warfarin. Level of evidence: "A"

Summary

A Cochrane review [Abstract] 1 included 8 studies with a total of 27 557 participants with non-valvular atrial fibrillation (AF) and one or more risk factors for stroke; 26 601 of them were assigned to standard doses groups and included in the primary analysis. Dabigatran 110 mg twice daily and 150 mg twice daily, AZD0837 300 mg once per day and ximelagatran 36 mg twice per day were compared with the vitamin K antagonists (VKA) warfarin with target INR 2.0 to 3.0. The mean age of participants in all studies was over 70 years, and 61% of participants were men. The rate of participants never previously exposed to VKAs was approximately 50% (range 5% to 100%). The average CHADS2 score, if reported, was 2.1 except for one study in which the average score was 3. In the groups assigned to warfarin, the INR was maintained within the therapeutic range between 57% and 71% of the time.

The odds of vascular death and ischaemic events were not significantly different between all direct thrombin inhibitors (DTIs) and warfarin (table T1). Sensitivity analysis by dose of dabigatran indicated that dabigatran 150 mg twice daily was superior to warfarin although the effect estimate was of borderline statistical significance.

Vascular deaths and ischaemic events* with DTIs versus warfarin

InterventionRelative effectControl (warfarin)Intervention (95% CI)Participants (studies)
Direct thrombin inhibitors (all)OR 0.94 (0.85 to 1.05)23 per 100022 per 1000 (20 to 24)26 601 (8 studies)
Dabigatran (all doses)OR 0.92 (0.82 to 1.04)74 per 100068 per 100 (61 to 77)18 509 (3 studies)
Dabigatran 150 mg twice dailyOR 0.86 (0.75 to 0.99)74 per 100064 per 1000 (56 to 73)12 448 (3 studies)
* Vascular deaths and ischaemic events includes non-fatal ischaemic strokes and TIAs, non-fatal systemic embolic events (any event of acute non-intracerebral or non-coronary vascular origin including deep vein thrombosis and pulmonary embolism), non-fatal myocardial infarction, any death related to a vascular cause not including fatal haemorrhages or cardiovascular deaths (e.g. sudden arrhythmia, pump failure).
Fatal and non-fatal major bleeding events, including haemorrhagic strokes, were less frequent with all DTIs compared with warfarin, but no difference was observed between dabigatran 150 mg and warfarin (table T2). Adverse events other than bleeding and ischaemic events that led to treatment discontinuation were significantly more frequent with all DTIs (OR 2.18, 95% CI 1.82 to 2.61; 5 studies, n=19143) and dabigatran (OR 2.12, 95% CI 1.77 to 2.56; 3 studies, n=18 509) compared with warfarin. Serious adverse events were also more frequent with all DTIs (OR 1.31, 95% CI 1.09 to 1.56; 5 studies, n=19 077) and dabigatran (OR 1.35, 95% CI 1.12 to 1.63; 3 studies, n=18 443) than warfarin. There was no difference in all-cause mortality between all DTIs (OR 0.91, 95% CI 0.83 to 1.01; 8 studies, n=26 601), or dabigatran (OR 0.90, 95% CI 0.80 to 1.01; 3 studies, n=18 509) and warfarin.

Fatal and non-fatal haemorrhages with DTIs versus warfarin

InterventionRelative effectControl (warfarin)Intervention (95% CI)Participants (studies)
Direct thrombin inhibitors (all)OR 0.87 (0.78 to 0.97)39 per 100034 per 1000 (31 to 38)26 601 (8 studies)
Dabigatran (all doses)OR 0.92 (0.82 to 1.03)79 per 100073 per 1000 (66 to 81)18 509 (3 studies)
Dabigatran 150 mg twice dailyOR 1.01 (0.89 to 1.16)79 per 100080 per 1000 (71 to 91)12 448 (3 studies)

Ximelagatran has been withdrawn from the market owing to toxic effects on the liver.

A systematic review 2 and meta-analysis included 7 retrospective cohort studies with a total of 348 750 subjects evaluating dabigatran compared to warfarin for stroke prevention in nonvalvular atrial fibrillation. There was no benefit of either dabigatran dose (150 mg or 110 mg) over warfarin in preventing ischemic stroke, but both dabigatran 150 mg (HR 0.44, 95% CI 0.34 to 0.59; statistical heterogeneity I2 =63.6%) and dabigatran 110 mg (HR 0.49, 95% CI 0.34 to 0.72) had lower hazards of intracranial bleeding compared to warfarin. Dabigatran 150 mg had a greater hazard of gastrointestinal bleeding compared to warfarin (HR 1.23, 95% CI 1.01 to 1.50; statistical heterogeneity I2 =89.8%), which was potentiated in studies of older versus younger populations (median/mean age, HASH(0x2fd57c0)75 versus <75 years).

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    References

    • Salazar CA, Del Aguila D, Cordova EG. Direct thrombin inhibitors versus vitamin K antagonists for preventing cerebral or systemic embolism in people with non-valvular atrial fibrillation. Cochrane Database Syst Rev 2014;(3):CD009893. [PubMed]
    • Romanelli RJ, Nolting L, Dolginsky M, et al. Dabigatran Versus Warfarin for Atrial Fibrillation in Real-World Clinical Practice: A Systematic Review and Meta-Analysis. Circ Cardiovasc Qual Outcomes 2016;9(2):126-34 [PubMed]

    Primary/Secondary Keywords