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Basics

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DESCRIPTION

FORMS AND USES

TOXIC DOSE

Ingestion of 1 to 2 mg/kg may result in hyperactivity and mydriasis in children.

PATHOPHYSIOLOGY

Methylphenidate overdose produces a hyperadrenergic state similar to that induced by amphetamines.

EPIDEMIOLOGY

CAUSES

DRUG AND DISEASE INTERACTIONS

PREGNANCY AND LACTATION


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Diagnosis

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DIFFERENTIAL DIAGNOSIS

SIGNS AND SYMPTOMS

Acute intoxication is characterized by agitation, tachycardia, hypertension, and mydriasis.

Vital Signs

Tachycardia, hypertension, and hyperthermia occur with moderate to severe overdose.

HEENT

Dermatologic

Diaphoresis is common early in the course of moderate to severe overdose.

Pulmonary

Cardiovascular

Gastrointestinal

Nausea, vomiting, anorexia, and abdominal pain may occur.

Hepatic

Hepatitis occurs rarely.

Renal

Acute renal failure may result from dehydration, seizures, rhabdomyolysis, or hypotension.

Fluids and Electrolytes

Dehydration, hypokalemia, and lactic acidosis may occur.

Musculoskeletal

Agitation may lead to rhabdomyolysis.

Neurologic

PROCEDURES AND LABORATORY TESTS

Essential Tests

No tests may be needed in asymptomatic patients.

Recommended Tests

Not Recommended Tests

Serum methylphenidate levels are not clinically useful.


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Treatment

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DIRECTING PATIENT COURSE

The health-care provider should call a poison control center when:

The patient should be referred to a health-care facility when:

Admission Considerations

Inpatient treatment is warranted when patients present with refractory agitation, seizure, hyperthermia, persistent tachycardia, or other end-organ injury.

DECONTAMINATION

Out of Hospital

Induction of emesis is not recommended due to seizure potential.

In Hospital

ANTIDOTES

There are no specific antidotes for methylphenidate poisoning.

ADJUNCTIVE TREATMENT

Agitation

Seizures

Hypertension

If hypertension persists after treatment of agitation with benzodiazepine or end-organ damage develops (e.g., aortic dissection, central nervous system bleed, myocardial infarction), a short-acting titratable agent such as nitroprusside should be administered.

Hypotension

Ventricular Dysrhythmia

See SECTION II, Ventricular Dysrhythmias chapter.

Rhabdomyolysis

Adequate hydration and urine output (1-2 ml/kg/h) should be ensured. Urinary alkalinization may be beneficial, but definitive data are not available.

Not Recommended Therapies

beta-blocker therapy for tachycardias or hypertension may result in unopposed alpha-adrenergic receptor stimulation and worsening of hypertension.


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FollowUp

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PATIENT MONITORING

ECG, respiratory and hemodynamic function, and core temperature should be monitored.

EXPECTED COURSE AND PROGNOSIS

DISCHARGE CRITERIA/INSTRUCTIONS


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Pitfalls

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DIAGNOSIS

TREATMENT

Intravenous abusers are at risk for the complications of intravenous drug use.


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Miscellaneous

ICD-9-CM 969.7

Poisoning by psychotropic agents: psychostimulants.

See Also: SECTION II, Hypotension, Seizures, and Ventricular Dysrhythmias chapters; and SECTION III, Nitroprusside and Whole-Bowel Irrigation chapters.

RECOMMENDED READING

Sherman CB, Hudson LD, Pierson DJ. Severe precocious emphysema in intravenous methylphenidate (Ritalin) abusers. Chest 1987;92:1085-1087.

Stecyk O, Loludice TA, Demeter S, et al. Multiple organ failure resulting from intravenous abuse of methylphenidate hydrochloride. Ann Emerg Med 1985;14:597-599.

Authors: Lada Kokan and Steven A. Seifert

Reviewer: Luke Yip